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Based on reading some of the websites it seems that their hypothesis is that because ALC is involved in the Krebs cycle in the mitochondria, which gets energy from fatty acids, that means ALC helps burn fat. There is not much in the scientific literature on this topic, but I did come across an article that found no difference in metabolic parameters in study participants taking L-carnitine versus controls (not exactly the same things as acetyl-L-carnitine, but they are used somewhat similarly in the body, there just isnt the extra acetyl group in the L-carnitine)

The Results In this study, 3,346 people with obesity, meaning a BMI over 30, but without diabetes started GLP-1 therapy and were compared to 8,343 similar people not using GLP-1 drugs

GIP-(342) does not antagonize insulinotropic effects of GIP at physiological concentrations

1415456-99-3 PubChem CID 167312356 Source Synthetic Form Powder Description Cagrilintide is a non-selective AMYR/CTR agonist and long-acting acylated amylase analogue