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Cartilage, Cardiac & Mechanical Stress Contexts Further experimental work suggests that MGF IGF-1 EC may assist cells in cartilage and cardiac tissue to resist mechanical stress, apoptosis or ischemia
Finally, this growth advantage translated to faster growth of GGT1 + cells in vivo (Fig
M1 secretes pro-inflammatory cytokines (mainly IL-1, IL-6, and TNF), which lead to osteoclast (OC) activation and joint erosion, and M2 produces anti-inflammatory cytokines such as IL-10 and transforming growth factor- (TGF-) to promote angiogenesis and tissue remodeling [82, 83]
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